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Vendor: Cisco Exam Code: 200-125 Exam Name: CCNA Cisco Certified Network Associate CCNA (v3.0) Version: DemoDEMO

QUESTION 1 A network administrator needs to configure a serial link between the main office and a remote location. The router at the remote office is a non-Cisco 200-125 dumps router. How should the network administrator configure the serial interface of the main office router to make the connection? A. Main(config)# interface serial 0/0 Main(config-if)# ip address Main(config-if)# no shut B. Main(config)# interface serial 0/0 Main(config-if)# ip address Main(config-if)# encapsulation ppp Main(config-if)# no shut C. Main(config)# interface serial 0/0 Main(config-if)# ip address Main(config-if)# encapsulation frame-relay Main(config-if)# authentication chap Main(config-if)# no shut D. Main(config)# interface serial 0/0 Main(config-if)#ip address Main(config-if)#encapsulation ietf Main(config-if)# no shut Correct Answer: B

QUESTION 2 Which Layer 2 protocol encapsulation type supports synchronous and asynchronous circuits and has built- in security mechanisms? A. HDLC B. PPP C. X.25 D. Frame Relay Correct Answer: B

QUESTION 3 Refer to the exhibit. The two connected ports cissp dumps on the switch are not turning orange or green. What would be the most effective steps to troubleshoot this physical layer problem? (Choose three.)A. Ensure that the Ethernet encapsulations match on the interconnected router and switch ports. B. Ensure that cables A and B are straight-through cables. C. Ensure cable A is plugged into a trunk port. D. Ensure the switch has power. E. Reboot all of the devices. F. Reseat all cables. Correct Answer: BDF

QUESTION 4 A network administrator is troubleshooting the OSPF 100-105 dumps configuration of routers R1 and R2. The routers cannot establish an adjacency relationship on their common Ethernet link. The graphic shows the output of the show ip ospf interface e0 command for routers R1 and R2. Based on the information in the graphic, what is the cause of this problem? A. The OSPF area is not configured properly. B. The priority on R1 should be set higher. C. The cost on R1 should be set higher. D. The hello and dead timers are not configured properly. E. A backup designated router needs to be added to the network. F. The OSPF process ID numbers must match. Correct Answer: D

QUESTION 5 Standard industrialized protocol of etherchannel? A. LACP B. PAGP C. PRP D. REP Correct Answer: A

210-260 dumps QUESTION 6 Two features of the extended ping command? (Choose two.) A. It can send a specific number of packet B. It can send packet from specified interface of IP address C. It can resolve the destination host nameD. It can ping multiple host at the same time Correct Answer: AB

QUESTION 7 What command is used to configure a switch as authoritative NTP server? A. ntp master 3 B. ntp peer IP C. ntp server IP D. ntp source IP Correct Answer: A

QUESTION 8 Two statements about syslog loging? A. Syslog logging is disabled by default B. Messages are stored in the internal memory of device C. Messages can be erased when device reboots D. Messages are stored external to the device E. other F. other Correct Answer: AD

QUESTION 9 How to enable vlans automatically across multiple switches? A. Configure VLAN B. Confiture NTP C. Configure each VLAN D. Configure VTP Correct Answer: D

QUESTION 10 Refer to the exhibit. A network administrator is configuring an EtherChannel 300-101 dumps between SW1 and SW2. The SW1 configuration isshown. What is the correct configuration for SW2? A. interface FastEthernet 0/1 channel-group 1 mode active switchport trunk encapsulation dot1q switchport mode trunk interface FastEthernet 0/2 channel-group 1 mode active switchport trunk encapsulation dot1q switchport mode trunk B. interface FastEthernet 0/1 channel-group 2 mode auto switchport trunk encapsulation dot1q switchport mode trunk interface FastEthernet 0/2 channel-group 2 mode auto switchport trunk encapsulation dot1q switchport mode trunk C. interface FastEthernet 0/1 channel-group 1 mode desirable switchport trunk encapsulation dot1q switchport mode trunk interface FastEthernet 0/2 channel-group 1 mode 640-911 dumps desirable switchport trunk encapsulation dot1q switchport mode trunk D. interface FastEthernet 0/1 channel-group 1 mode passive switchport trunk encapsulation dot1q switchport mode trunk interface FastEthernet 0/2 channel-group 1 mode passive switchport trunk encapsulation dot1q switchport mode trunk Correct Answer: C

300-135 400-051 101 adm-201 1z0-808 CCA-500 1v0-621 mb2-707 70-980 70-483 2v0-621 nse4 1z0-434 9l0-012 101-400 300-085 og0-093 1z0-061 70-488 1z0-062 mb5-705 102-400 PEGACPBA71V1 70-463 mb2-704 PR000041 IIA-CIA-PART1 700-037 PEGACSA71V1 1z0-144 2v0-621d 1z0-051 070-461 a00-211 jn0-102 1z0-804 640-875 API-580 3002 400-151 98-365 712-50 9l0-066 ns0-506 156-215.77 70-466 lx0-104 9a0-385 642-980 og0-091 74-678 700-260 70-494 c_tfin52_66 lx0-103 m70-101 pmi-001 DEV-401 1z0-067 1K0-001 220-801 TB0-123 700-038 IIA-CIA-PART2 cwna-106 070-487 hp0-y50 070-483 mb2-708 C2010-595 1z0-883 c_tadm51_731 pk0-003 700-039 jn0-633 98-364 300-080 74-343 1z0-133 70-465 c_tscm62_66 PRINCE2-PRACTITIONER mb6-704 1v0-605 API-571 500-007 and-401 c_taw12_731 AX0-100 070-463 70-981 1z0-052 070-488 c_hanatec_10 010-111 mb6-700 700-270 600-455 600-460 1z0-533

This is a correspondence from

Darren Orbach, MD PhD
Division Chief, Interventional & Neurointerventional Radiology
Children’s Hospital Boston / Harvard Medical School
300 Longwood Avenue
Boston, MA 02115

Hi Mr. Christou,

Thanks for your email. As someone who treats both brain/spine AVMs and extracranial AVMs, I would certainly concur with your assessment that in some ways treating the peripheral lesions can be more challenging than treating the brain lesions. In particular, with most brain AVMs we treat, we think in terms of cure – i.e. getting the patient to a state whereby the risk of the AVM posing a clinical challenge to the patient is minimal. Unfortunately, it is only for the tiniest minority of peripheral AVMs that we can talk of cure in this way – for the vast majority, our goal is to control symptoms and minimize the impact of the AVM on the patient’s day-to-day life. Part of this distinction is due to the fact that brain AVMs pose an ongoing risk to the patient’s life, in a way that is rarely true for peripheral AVMs, so the treatment approaches that have developed through the years for brain AVMs have been oriented towards cure, whenever possible for many years. But a big part of the reason for the distinction is technical – brain AVMs are more distinct from their surrounding tissue, both in their microarchitecture, and visually to the surgeon, than are peripheral AVMs.
Thus, my neurosurgical colleagues who resect brain tumors that I have embolized usually have little trouble distinguishing AVM from brain tissue. In contrast, for the few patients we send to the OR to have the plastic surgeons resect their peripheral AVMs, it is rarely easy, or even possible, to distinguish where in the soft tissue the AVM ends and the normal muscle or fat tissue begins.

Because of this distinction, our approach towards peripheral AVMs is to treat them when they are symptomatic. Most often, this is by way of embolization. I do happen to use Onyx a great deal, both in the CNS and in the periphery, and tend to favor it because of the exquisite control it offers the operator in terms of target deposition. However, Onyx is a tool just like the other liquid embolic agents, and I’m not convinced that using it really changes the natural history of peripheral AVMs (or at least, I’ll readily admit the that the jury is still out). I have not found the black color of Onyx to be problematic, as most AVMs are sufficiently below the skin surface that there is at most minimal discoloration – when visible at all, the appearance is similar to the blueish appearance of a vein under the skin. In cases where Onyx is likely to cause significant discoloration, the lesion itself is so superficial, that the skin has already darkened significantly. I have had a few cases where I was concerned enough about discoloration that I used n-BCA rather than Onyx, but these are the exception.

There is certainly a lot of research ongoing with regard to pharmacological approaches to AVM, and given the pretty rapid development of our understanding of the underlying vascular biology, I’m confident that 10 years from now, we will have far superior active agents to offer our patients, either by mouth or by directed infusions into the lesion. For cases on which we at the Children’s Vascular Anomalies Center are consulted, where there is a lesion that is both dangerous and that cannot be managed by standard means, we do offer trials of angiogenesis-inhibiting medications.

However, these are still early in their development and have no accompanying long-term data with regard to efficacy and safety, and are not something to which we would point patients whose lesions can be better managed by more standard approaches.

I hope all this helps, and best of luck to you and your daughter.

Best regards,

-Darren Orbach